No inflammatory cell infiltration and fibrous hyperplasia.
未见炎性细胞浸润和纤维增生。
Around adnexal inflammatory cell infiltration is also evident.
附属器周围亦有明显炎细胞浸润。
Intensive inflammatory cell infiltration and fibroblast hyperplasia were presented.
大部分肺泡腔及肺泡管内透明膜形成。间质有较多的炎细胞浸润，成纤维细胞增生。
The changes mainly were edema, hyperemia and inflammatory cell infiltration.
主要病理改变为水肿，炎症细胞浸润和充血。
The changes mainly were edema, hyperemia an d inflammatory cell infiltration.
病变部位可以出现在粘膜层，粘膜下层，肌层和浆膜层。
Slight edema and inflammatory cell infiltration in the cerebella were observed.
小脑组织轻度水肿，有少量炎性细胞浸润，神经细胞正常，神经胶质细胞轻微增生；
Inflammatory cell infiltration can be spread to the subcutaneous adipose tissue.
炎细胞浸润可波及到皮下脂肪组织。
Inflammatory cell infiltration and index of remodeling of bronchiole were detected.
测细支气管炎症细胞浸润及支气管重塑指标；
Enema with low dose group compared with saline solution group, lamina propria layer in a small number of chronic inflammatory cell infiltration, submucosa edema, telangiectasias, small amounts of chronic inflammatory cell infiltration.
低剂量灌肠组与生理盐水组相比，粘膜固有层散在少量慢性炎细胞浸润，粘膜下层水肿，毛细血管扩张，少量慢性炎细胞浸润。
There was also an interstitial subacute inflammatory cell infiltration associated with the lipid deposition.
脂质沉积使间质亚急性炎症细胞渗透。
RESULTS The histological responses consisted of inflammatory cell infiltration, necrosis, vasal responses and fibroplasia.
结果组织学反应以炎细胞浸润、坏死、血管反应及纤维组织增生为主。
Results: Inflammatory cell infiltration and phagocyte cell apoptosis were observed in discolored gingiva with TEM.
结果：透射电镜下观察到组织中炎症细胞浸润，吞噬细胞增多，其细胞器发生改变及凋亡的早期表现；
Light only see a small amount of inflammatory cell infiltration. In more severe cases, it can be seen that a large number of inflammatory cell infiltration with fibrous tissue hyperplasia and obvious organize congestion.
轻者仅见少量炎性细胞浸润；重者可见大量炎性细胞浸润，纤维组织增生，组织充血。
Pathological histology is mainly expressed in fatty degeneration of liver cell injury and inflammatory cell infiltration.
病理组织学主要表现为脂肪变性、肝细胞损伤及炎症细胞浸润。
Inflammatory cell infiltration was observed in the cochlear nerve and the number of spiral ganglion cells detected.
均有不同程度的螺旋神经节细胞变性、数目减少，螺旋神经节和小血管周围有淋巴细胞浸润；
Conclusions EAE is characterized by perivascular inflammatory cell infiltration and demyelination in white matter and some remyelination.
结论EAE的病理改变为血管周围炎性细胞浸润、白质脱髓鞘及髓鞘重建。
Results After hyperoxia exposure, there were edema and inflammatory cell infiltration in the lung tissues of SD rats.
结果高氧暴露的SD大鼠肺组织可见水肿、大量炎症细胞浸润等急性炎症反应。
Results Faster corneal epithelial healing, less corneal neovascularization and inflammatory cell infiltration occurred in the animal operative group.
结果动物手术组角膜上皮愈合快，角膜新生血管及炎细胞浸润均轻于对照组。
Results Under light microscope, degenerative atrophy and demyelination were shown in nerve fibers without inflammatory cell infiltration in stroma;
结果光镜下见神经纤维变性萎缩，部分区域呈现脱髓鞘改变，间质中未见炎症细胞浸润；
Result: Tongbiling could inhibit inflammatory cell infiltration and fibroblast hyperplasia and macrophage like type A synoviocyte hyperplasia in CIA rats.
结果：通痹灵对CIA大鼠滑膜组织的炎性细胞浸润、纤维组织增生和巨噬样A型细胞有明显抑制作用。
Inflammatory cell infiltration closely correlated with the loss of elas-tin and collagen fiber during the whole processes of AAA.
炎性细胞浸润与弹力纤维和胶原纤维的损伤密切相关，这个过程贯穿于AAA形成的始终。
Both high and low group could significantly reduce the inflammatory cell infiltration in rats prostatic tissue, inhibit fibrous connective tissue proliferation.
高、低剂量组均可明显减轻模型大鼠前列腺组织内炎细胞浸润，抑制纤维结缔组织增生。
The pathological features of autoimmune hepatitis is inflammatory cell infiltration in the portal area and invading to the surrounding liver parenchyma.
自身免疫性肝炎的病理特征为大量炎性细胞浸润于汇管区，并向周围的肝实质侵犯。
On pathological findings, that congestion and inflammatory cell infiltration in the walls of intestine and colon as well as mesenterium were found.
组织病理学检查发现肠系膜及肠壁血管充血、炎性细胞浸润。
After oral administration, BPs significantly improved pathological injury. Inflammation were reduced, alveolar hemorrhage were extenuate, alveolar wall and inflammatory cell infiltration were decreased.
口服给药后，柴胡明显改善了病理损伤，病理切片显示给药后炎症程度明显减轻，肺泡腔出血明显减少，肺泡壁变薄，炎性细胞浸润减少。
